In pharmacokinetic characterization, the DAC-modified peptide had a plasma half-life of roughly 8 days — extending its circulating lifetime from minutes to more than a week.
Reported half-life after albumin binding.
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Long-acting GHRH analogue, supplied for laboratory research use only.
$79/ 5mg vial
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All products currently listed on this site are for research purposes only.
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A long-acting growth-hormone-releasing research peptide studied across the GH–IGF-1 axis.
Studied for binding the growth-hormone-releasing-hormone receptor to prompt GH secretion.
Investigated for holding growth-hormone levels elevated over multi-day windows via albumin binding.
Researched for the downstream rise in IGF-1 across the GH axis.
For research use only. Mechanisms described reflect published research literature.
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Published findings on this compound, each linked to its source. Animal and lab results are context, not claims about this product.
In pharmacokinetic characterization, the DAC-modified peptide had a plasma half-life of roughly 8 days — extending its circulating lifetime from minutes to more than a week.
Reported half-life after albumin binding.
In preclinical work, GH and IGF-1 stayed elevated across a multi-day window rather than dropping back to baseline between pulses — the kind of steady signal researchers want for studying the GH–IGF-1 axis.
Directional; the sustained arm holds elevation between pulses.
Reviewers attribute the long duration to the Drug Affinity Complex (DAC), which binds serum albumin to slow the body's clearance of the peptide versus the unmodified form.
Mechanistic; relative circulation time, not a measured value.
Quick answers about CJC-1295 with DAC.
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